Categories
Lyme disease

Guinea Fowl Prevent Lyme Disease by Reportedly Eating up to 4000 Ticks per Day

Known by a few names such as African Pheasant and Guinea Hens, one could also call this African native the “Tick Assassin.”

In their homeland, these loud and prehistoric-looking fowl follow around the grazing herds and reportedly eat up to 4000 ticks a day.

Guinea fowl are relatively low maintenance. In the summertime, guinea will forage all of their food. They eat ticks, fleas, crickets, mosquitos, slugs, grasshoppers and small rodents (basically anything they can get their beaks on) without destroying the yard or garden as chickens do. Flocks of guineas have been known to eat snakes. They also love to eat weeds. A bit of feed in the coop will encourage them to come home at night. Coops can be provided, although flocks will find homes high in treetops, away from the threat of raccoons.

They are also free from most diseases that plague other poultry. They will give warning calls when unknown guests arrive, which helps to protect other farm animals. Their call will also keep unwanted rodents away.

Guinea fowl manure can be added to your compost and is a wonderful addition to nurturing the soil where your food is grown. Your plants will thrive.

Guineas are known for their hardiness, being able to withstand both hot and cold.

While not laying as often as chickens, guinea eggs may still be collected and eaten. Guineas tend to make their nests in hidden areas, so finding their eggs may be an adventure.

When starting your flock, buy your keets from a reputable source.

When starting your guinea flock, it’s best to buy them from keets (babies) so they will know where home is. Imported adult guineas will wander off.

If you have nearby neighbors, it’s best to check in with them first. Their squawking can be unbearable for some. However, given their reputation as tick assassins, having them in the neighborhood can be a helpful, healthy alternative to pesticides if you are living in a tick-ridden area.

Bear in mind that guinea meat is also a known delicacy, so if you start your flock and someone complains, you can also have guinea stew.

Categories
Celiac disease

NANOPARTICLE TECH REDUCES CELIAC DISEASE SYMPTOMS BY 90%

People with celiac disease have two options in life, neither of which is ideal.

Because their immune systems can’t tolerate gluten, they can choose to never eat the many delicious foods containing it. Boring.

Or they can devour all the cake, bread, and beer they want — but resign themselves to abdominal pain, diarrhea, and other nasty side effects when their immune systems trigger an inflammation response in their small intestines.

Needless to say, people tend to choose the former option — but a new technology could allow them to have their cake and feel good about the decision later, too.

Researchers from Northwestern University developed the tech, which they presented on Tuesday at the European Gastroenterology Week conference, and it works by hiding a bit of gluten in a biodegradable nanoparticle.

Injected into a person’s bloodstream, the nanoparticle looks to the immune system like a bit harmless debris, so it allows a macrophage — a type of cell tasked with removing such detritus from the body — to gobble up the particle, hidden gluten and all.

“The vacuum-cleaner cell presents the allergen or antigen to the immune system in a way that says, ‘No worries, this belongs here,’” researcher Stephen Miller said in a news release. “The immune system then shuts down its attack on the allergen, and the immune system is reset to normal.”

What’s exciting is that the researchers have already tested the nanoparticle in a phase 2 clinical trial of people with celiac disease.

They gave some participants two intravenous treatments of their nanoparticle tech and others nothing to serve as a control group. A week later, they fed both groups gluten for 14 straight days and found the treated group experienced 90 percent less immune inflammation than participants in the control group.

So far, the researchers have only tested their system in humans to combat celiac disease, but they believe they could eventually treat peanut allergies, multiple sclerosis, type 1 diabetes, and a host of other issues all with the same Trojan horse-style tech.

Categories
Lyme disease

20 Surprising Symptoms of Lyme Disease You Can’t Afford to Ignore

FROM EXTREME FATIGUE TO SWOLLEN FEET, THESE ARE THE DIVERSE SYMPTOMS OF THE COMPLEX ILLNESS.

When people think about Lyme disease, what immediately comes to mind is the so-called bullseye rash that’s most often associated with the tick-borne illness. However, according to the Centers for Disease Control and Prevention (CDC), this erythema migrans rash only occurs in an estimated 70 to 80 percent of Lyme patients, which means that as many as 30 percent of those with the disease must rely on other symptoms in order to get a proper diagnosis. But unlike other conditions that have telltale signs and symptoms, Lyme disease is all over the map when it comes to how it manifests, making it especially difficult to diagnose.

“It is quite a complicated infection. There are a lot of non-specific systems—and that’s part of the problem,” explains Kenneth Liegner, MD, a New York-based board-certified internist who’s been involved with Lyme disease research since 1988. “Anybody who thinks it’s all cut and dry… that’s definitely not true.”

So, what can you do to make sure that you aren’t infected? Familiarize yourself with these surprising—though not uncommon—Lyme disease symptoms you can’t afford to overlook.

1. Sore throat

One reason why doctors have so much difficulty diagnosing individuals with Lyme disease is because of how often the illness manifests as a sore throat. A 2011 study about the similarities between Lyme and other summer illnesses published in the journal Orthopedic Reviews notes that “respiratory symptoms such as sore throat may occur in non-viral summer infections such as Lyme disease.”

2. Jaw pain

Does it feel like your head is about to explode every time you chew? Well, this too could be a sign that you have Lyme disease. The Lyme Disease Association in New Jersey notes that TMJ—short for temporomandibular joint dysfunction—is one of the many ways that this tick-borne illness can present itself in patients.

3. Trouble sleeping

Whether you’ve just contracted Lyme disease or have unknowingly had it months, odds are that you’re having trouble sleeping. According to LymeDisease.org, approximately 41 percent of people with early-stage Lyme disease have sleep issues, while 66 percent of chronic Lyme patients do.

4. Extreme fatigue

 

It’s normal to feel fatigued after a long day at work. What’s not normal is getting nine hours of uninterrupted sleep only to wake up and feel like someone kept you up all night blasting music. If you find that no amount of sleep is doing the trick for you, Tufts University School of Medicine professor Linden Hu, MD, notes that this could be a sign of Lyme disease—one that could potentially linger for months after being treated.

5. Headaches

If you’re worried that you might have contracted Lyme disease, then make sure to monitor the frequency of your headaches. According to the CDC, one of the early signs of Lyme disease that tends to occur within the first 30 days of a tick bite is head pain.

And one 2003 study published in the journal Pediatrics detailed two cases of Lyme disease in which patients presented with headaches. The researchers concluded that “it is important for practitioners to consider Lyme disease when patients present with persistent headache,” particularly in areas where the disease is common.

6. Joint pain

You shouldn’t assume that you have regular age-related arthritis just because you’re in your 50s or 60s. Rather, the CDC notes that joint pain is one of the more surprising symptoms of late-stage Lyme disease. According to one 2013 study published in the Journal of the American Academy of Orthopaedic Surgeons, as many as 60 percent of untreated patients will experience so-called Lyme arthritis.

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Categories
Parkinson's Disease

8 Common Treatments for Parkinson’s Disease

Parkinson’s disease is a long-term degenerative disease that affects the central nervous system. To date, there is no cure for the condition, but there are medications and therapies available to address some of the symptoms and improve quality of life for patients.

Here are eight of the main drugs and therapies used in the treatment of Parkinson’s disease, according to the Mayo Clinic:

1. Carbidopa-levodopa: Levodopa is a naturally occurring chemical which can enter the brain and be converted to dopamine when combined with carbidopa. The carbidopa also prevents the levodopa from converting into dopamine before it enters the brain.

The is one of the most effective treatments for Parkinson’s although after long-term use, the effects start to fluctuate.

Some people may experience side effects such as nausea, feeling lightheaded, and making sudden involuntary movements.

2. Carbidopa-levodopa infusion: In 2015, the FDA approved Duopa, which is a combination of carbidopa and levodopa in a gel form which is administered via a feeding tube into the small intestine.

MORE: Parkinson’s Disease and Neuroplasticity

Duopa is generally given to patients with advanced Parkinson’s disease whose response to carbidopa-levodopa is varied. The drug is infused continuously so the level of the drugs remains constant.

The risks associated with Duopa are infections at the site of the feeding tube and the tube falling out.

3. Dopamine agonists: Dopamine agonist mimic the effects of dopamine in the brain. They are generally not as effective as levodopa but the effects last longer and they can be used in conjunction with levodopa to counter any fluctuation in efficiency.

These medications can be administered through a patch, oral medications or as an injection. The side effects are also nausea and lightheadedness, but may also cause drowsiness, hallucinations and compulsive behaviors such as gambling, overeating, and hypersexuality — which will need to be addressed by a doctor.

4. MAO-B inhinitors: Medications such as selegiline and rasagiline help to prevent dopamine breaking down in the brain by releasing monoamine oxidase B (MAO-B) enzymes.

Generally, these types of medications should not be taken in conjunction with certain narcotics or antidepressants as occasionally patients will suffer from severe reactions. Side effects of MAO-B inhibitors include insomnia and nausea and if taken with carbidopa-levodopa they can also cause hallucinations.

 

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Categories
Parkinson's Disease

Parkinson’s: How stem cells can help repair the brain

New research examines the potential of stem cell therapy in the replacement of damaged neurons in Parkinson’s disease. The authors say that stem cells could “provide superior treatment, possibly using different types of cells to treat different symptoms” of Parkinson’s.

scientist working in lab
Could stem cell therapy one day help treat Parkinson’s disease?

Parkinson’s disease affects around half a million people in the United States, and the number is only expected to rise given the progressive aging of the population.

The National Institutes of Health (NIH) estimate that doctors diagnose the condition in around 50,000 people each year.

The NIH warn that the prevalence of this neurodegenerative condition is only going to increase unless researchers come up with new and better treatments.

Currently, the most common therapy uses the drug levodopa to stimulate dopamine production in certain neurons associated with motor skills.

These dopaminergic neurons are situated in the nigrostriatal pathway, which is a brain circuit that connects neurons in the substantia nigra pars compacta with the dorsal striatum.

However, levodopa has a wide array of side effects, from physiological to psychological ones. Also, in the long-term, the benefits of such dopamine-regulating drugs are limited.

So, it is crucial that scientists come up with more effective strategies for repairing the brain damage that Parkinson’s disease causes.

New research, which now appears in a special supplement to the Journal of Parkinson’s Disease, evaluates the potential of stem cell therapy for treating this neurodegenerative condition.

Dr. Claire Henchcliffe, of the Department of Neurology at the Weill Cornell Medical College in New York City, NY, co-authored the study with Malin Parmar, Ph.D., a professor in a research group called “Multidisciplinary research focused on Parkinson’s disease at Lund University.”

“We are in desperate need of a better way of helping people with [Parkinson’s],” say the scientists. “It is on the increase worldwide. There is still no cure, and medications only go part way to fully treat[ing] incoordination and movement problems.”

Stem cell therapy: Challenges and promises

In their review, Dr. Henchcliffe and Prof. Parmar examined the evolution of stem cell therapy and its uses for replacing damaged neurons in Parkinson’s.

“If successful, using stem cells as a source of transplantable dopamine-producing nerve cells could revolutionize care of the [Parkinson’s] patient in the future,” they say.

“A single surgery,” the authors go on to state, “could potentially provide a transplant that would last throughout a patient’s lifespan, reducing or altogether avoiding the need for dopamine-based medications.”

More than 3 decades ago, pioneering studies that transplanted stem cells to treat Parkinson’s used “fetal cells obtained from the midbrain of aborted embryos.”

However, there were numerous ethical issues with the procedure, as well as a host of side effects. These included transplant rejection and involuntary movements called dyskinesias.

Recent advances in stem cell technology mean that the materials from which stem cells are derived are different and varied. For instance, researchers can use a person’s own skin to collect pluripotent cells and reprogram them directly into neuronal cells.

Cells can also be reprogrammed directly in the brain by injecting the conversion genes instead of the human skin cells. Researchers can also derive stem cells from the person’s own blood.

“We are moving into a very exciting era for stem cell therapy,” Prof. Parmar points out. “The first-generation cells are now being trialed and new advances in stem cell biology and genetic engineering promise even better cells and therapies in the future.”

As Dr. Henchcliffe says, “Right now, we are just talking about the first logical step in using cell therapies in [Parkinson’s].

Importantly, it could open the way to being able to engineer the cells to provide superior treatment, possibly using different types of cells to treat different symptoms of [Parkinson’s] like movement problems and memory loss.”

Dr. Claire Henchcliffe

Prof. Parmar goes on to point out that “[t]here is a long road ahead in demonstrating how well stem cell-based reparative therapies will work, and much to understand about what, where, and how to deliver the cells, and to whom.”

She concludes, “But the massive strides in technology over recent years make it tempting to speculate that cell replacement may play an increasing role in alleviating at least the motor symptoms, if not others, in the decades to come.”

Categories
Parkinson's Disease

Parkinson’s Disease and Neuroplasticity

Prior to 20 or so years ago, the brain was thought to be rigid in many respects. The saying “you can’t teach an old dog new tricks” is an example of this thinking. Perhaps now it should be “use it or lose it”!

parkinson's disease and neuroplasticityWe now know, that, through the science of Neuroplasticity, the brain has the natural ability to reorganize itself by forming new neural pathways and connections and is capable of change even after childhood, on into maturity, and even old age. Brain reorganization occurs by forming new neural pathways to bring about a needed function. This is put in place in the brain by mechanisms such as “axonal sprouting”, where undamaged axons grow new nerve endings to reconnect neurons whose links were severed or impaired. Neuroplasticity also means undamaged axons can also grow nerve endings to connect with other undamaged nerve cells. For example, if damage is done in one hemisphere of the brain the other undamaged hemisphere may take over some of its functions. This is achieved by stimulating the neurons through certain activities, like Ageless Grace, where the brain compensates for damage by forming new communications between intact neurons.

This discovery has enormous implications for the Parkinson’s community.

Parkinson’s disease symptoms include physical, emotional and brain issues – Ageless Grace meets all the requirements of a highly effective Neuroplasticity model and is ideally suited to Parkinson’s disease as it addresses the 5 functions of the Brain and all the functions of healthy aging in the 21 tools. Ageless Grace addresses kinesthetic learning as well as Cognitive, Dementia and Apathy issues.

To know that a Parkinson’s disease patient has some hope, and that symptoms and progression may be helped by rewiring the brain, can be life changing.

By practicing Ageless Grace, a Parkinson’s disease patient can enhance attention span, increase levels of working memory, speed up the brain’s processing power and thereby stimulate healing and improve poor balance and other movement disorders. Studies have shown that learning to tango can use the brains natural plasticity to make positive changes and Ageless Grace combines all the aspects, like learning to tango, necessary for Neuroplasticity to take place and is therefore a perfect model for Parkinson’s disease.

Neuroplasticity Benefits of Ageless Grace for Parkinson’s Disease. Here are a few examples:

DYSTONIA, RIGIDITY AND POSTURAL INSTABILITY

Dystonia is a neurological movement disorder, common to Parkinson’s disease in which sustained muscle contractions cause twisting, twitching and repetitive movements or abnormal postures. Muscles tend to be rigid and treatment is difficult and has been limited to minimizing the symptoms of these disorders usually with medications. Tool #1 Juicy Joints; Tool # 18 Shake it up Baby; #15 Balancing Act: address and help to minimize these symptoms.

BRADYKINESIA

Bradykinesia refers to slowness of movement and is the most characteristic clinical feature of Parkinson’s disease. The slowness of movement is most clear when initiating and executing actions or activities that require several successive steps and require fine motor control. In Ageless Grace, Tools #2, #4, #11, #13 #17 and #19, have both relatively fast movements while others are slower, requiring fine motor control – all access muscle memory formed in youth, maximizing neuroplasticity potential.

MICROGRAPHIA

Micrographia means “small writing.” It is a common symptom of Parkinson’s disease, which affects many voluntary and involuntary muscle movements through the loss of the brain chemical dopamine. #3 Spelling “B” addresses motor-function and kinesthetic learning associated with this Parkinson’s disease symptom while many tools such as Dance Party – Tool #21 are uplifting and thereby increase dopamine levels.

DEMENTIA & COGNITION

Right-left Brain Coordination and Hand- Eye Coordination are important in treating Parkinson’s disease and are found in many of the tools – in particular Tool #4 Front Row Orchestra, #6 Try Chi, #3 Spelling “B” and Tool #5 Zoo-ology, which all stimulate Cognitive Function, Memory, Recall and Imagination.

AKINESIA /APATHY

Akinesia is one of the classic symptoms of moderate to advanced Parkinson’s Disease, and manifests as temporary episodes of “freezing” during movement or difficulty in starting movement such as walking. Since the disorder progresses as a result of nerve damage that causes injury to the brain, the nervous system does not send any signals for making movement. The prevailing perception of Akinesia is that it is a consequence of dopamine depletion. However, some recent studies suggest that serotonin and norepinephrine are also depleted in people with Akinesia. Most Ageless Grace tools, if not all, address these motor symptoms and the hormone depletions and Parkinson’s disease patients who regularly practice Ageless Grace report relief and “happy,” “good’ and “relaxed” feelings- “natural highs” from the practice which raise the levels of these neurotransmitters and have huge Neuroplastic implications for the nervous system.

HYPOMIMIA

Hypomimia is also known as facial masking. It refers to the face being less expressive than usual or“looking blank”. Some of the symptoms are caused by the loss of so-called automatic movements. These include blinking and smiling; Exercise Tool #14 Saving Face is recommended to stimulate Facial Muscles, and relieve Headaches and TMJ, which are the result of constant tensing of facial muscles.

Conclusion: Neuroplasticity is a new science that has great potential for the future to help with social, psychiatric, personal, emotional and brain disease problems by recognizing that the brain is a plastic organ that can be retrained, grown, and repaired by using techniques such as Ageless Grace – well-suited to Parkinson’s disease and other Neurological diseases.

Categories
Rheumatoid arthritis

Rheumatoid arthritis – Diagnosis and treatment

Diagnosis

Rheumatoid arthritis can be difficult to diagnose in its early stages because the early signs and symptoms mimic those of many other diseases. There is no one blood test or physical finding to confirm the diagnosis.

During the physical exam, your doctor will check your joints for swelling, redness and warmth. He or she may also check your reflexes and muscle strength.

Blood tests

People with rheumatoid arthritis often have an elevated erythrocyte sedimentation rate (ESR, or sed rate) or C-reactive protein (CRP), which may indicate the presence of an inflammatory process in the body. Other common blood tests look for rheumatoid factor and anti-cyclic citrullinated peptide (anti-CCP) antibodies.

Imaging tests

Your doctor may recommend X-rays to help track the progression of rheumatoid arthritis in your joints over time. MRI and ultrasound tests can help your doctor judge the severity of the disease in your body.

Treatment

There is no cure for rheumatoid arthritis. But clinical studies indicate that remission of symptoms is more likely when treatment begins early with medications known as disease-modifying antirheumatic drugs (DMARDs).

Medications

The types of medications recommended by your doctor will depend on the severity of your symptoms and how long you’ve had rheumatoid arthritis.

  • NSAIDs. Nonsteroidal anti-inflammatory drugs (NSAIDs) can relieve pain and reduce inflammation. Over-the-counter NSAIDs include ibuprofen (Advil, Motrin IB) and naproxen sodium (Aleve). Stronger NSAIDs are available by prescription. Side effects may include stomach irritation, heart problems and kidney damage.
  • Steroids. Corticosteroid medications, such as prednisone, reduce inflammation and pain and slow joint damage. Side effects may include thinning of bones, weight gain and diabetes. Doctors often prescribe a corticosteroid to relieve acute symptoms, with the goal of gradually tapering off the medication.
  • Disease-modifying antirheumatic drugs (DMARDs). These drugs can slow the progression of rheumatoid arthritis and save the joints and other tissues from permanent damage. Common DMARDs include methotrexate (Trexall, Otrexup, others), leflunomide (Arava), hydroxychloroquine (Plaquenil) and sulfasalazine (Azulfidine).

    Side effects vary but may include liver damage, bone marrow suppression and severe lung infections.

  • Biologic agents. Also known as biologic response modifiers, this newer class of DMARDs includes abatacept (Orencia), adalimumab (Humira), anakinra (Kineret), baricitinib (Olumiant), certolizumab (Cimzia), etanercept (Enbrel), golimumab (Simponi), infliximab (Remicade), rituximab (Rituxan), sarilumab (Kevzara), tocilizumab (Actemra) and tofacitinib (Xeljanz).

    These drugs can target parts of the immune system that trigger inflammation that causes joint and tissue damage. These types of drugs also increase the risk of infections. In people with rheumatoid arthritis, higher doses of tofacitinib can increase the risk of blood clots in the lungs. Biologic DMARDs are usually most effective when paired with a nonbiologic DMARD, such as methotrexate.

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Categories
Osteoporosis

The truth about tequila and your bones(Tequila could reverse osteoporosis)

Tequila could reverse osteoporosis!

Drinking tequila is good for your bones!

Have a Third Margarita — Because Tequila’s Great for Your Bones!

Talk about misleading headlines! These are prime examples.

It’s true that a newly published study found that a substance in tequila (called agave tequilana, or tequila agave) might help maintain bone health. And that it could lead to new treatments for osteoporosis. But consider the details:

  • The study was performed on mice who had had their ovaries removed. This is by no means a perfect model for human osteoporosis.
  • The mice were treated with a type of agave tequilana, not tequila, for only eight weeks.
  • When compared with untreated mice, the treated mice were found to have larger thigh bones, and samples of their thigh bones contained more of a protein linked with bone growth (called osteocalcin). However, there was no long-term treatment with agave tequilana beyond the initial eight weeks, nor was there any assessment of whether this treatment would prevent osteoporosis.

The researchers suggested that sugars in the agave tequilana interacted with bacteria in the intestinal tracts of the mice to encourage absorption of minerals needed to build bones. So, a “healthy intestinal microbiome” may also be required for this approach to work.

What’s the catch?

I think this new research is intriguing. It’s entirely possible that certain types of agave (a plant that produces a honey-like nectar) could turn out to help people maintain or improve bone health. And considering the health impacts of osteoporosis — hip fractures, loss of mobility, and complications that can lead to death in some cases — such an advance can’t come too soon.

But any study in animals has to be considered highly preliminary. It’s simply unknown whether the results of this study apply to humans. In addition, the animals did not drink tequila. They were treated with a chemical found in tequila. So, the suggestion that we (humans) might improve our bone health by drinking margaritas is, in my view, just a way to grab attention. Even if we could fast forward a few years and confirm that agave tequilana improves human bone health, it’s unlikely that the treatment would be in the form of tequila.

Unfortunately, many people don’t read past the headlines. This is one time when that would be hazardous. The health impact of the alcohol in tequila — and the sugar content of agave — are just two of several “downsides” that could come about if you were worried about your bone health and took the headlines too literally.

Haven’t we been here before?

This new study on “tequila for osteoporosis” reminds me of past studies touting the health benefits of chocolate, wine, or coffee. The same week as the tequila story broke, other researchers reported that certain substances in red wine and coffee could improve cardiovascular health by changing the intestinal bacteria. Again, the study was in mice.

Claims that some of our favorite foods and drinks are actually good for us are not new. Some claims are better supported than others. For example, the evidence that coffee consumption may reduce the incidence of certain types of liver disease in humans is compelling. Still, it’s relatively rare that doctors actually “prescribe” these foods to prevent or treat disease. Perhaps they should. But, enthusiasm for doing so is tempered by concerns that excessive consumption may cause other, unhealthy effects.

Stand by

We’ll need much more research before tequila or anything in it can be recommended for bone health, or any other health concern. Until then, I hope medical writers — and readers — will be careful in how they interpret preliminary research. It’s one thing to hope that what you like is also good for you. It’s quite another for that to be any more than wishful thinking.

Source: health.harvard.edu

Categories
COPD

4 Effective Tips to Fight COPD: Treat it with Natural Remedies

COPD is the fourth leading cause of death in the United States(5). The term COPD stands for Chronic Obstructive Pulmonary Disease. It is a life-threatening health condition whose symptoms can be miserable for suffers; they include an excessive build-up of phlegm, shortness of breath, coughing and wheezing.

The primary causal factors of COPD are prolonged exposure to pollution and allergies, smoking, pre-existing health issues, and genetic inheritance. Here we have listed some effective natural treatments to effectively tackle the symptoms of COPD.

Herbs: Improve the Breathing Capacity of Lungs

Herbs are considered one of the safest ways to reduce or reverse damage to the lungs, reduce the symptoms associated with COPD, thwart persistent infections and enhance the body’s overall immunity. There are various kinds of herbs that work to enhance respiratory function and breathing.

Olive leaf works by reducing inflammation and treating underlying infections. It has antibacterial, antibiotic and anti-inflammatory properties. Cayenne pepper enhances the flow of blood and improves breathing. A mixture of honey, cayenne pepper, and apple cider vinegar can be extremely effective in treating COPD.  Other herbs that help relieve symptoms of COPD include ginseng, milk thistle, thyme, Echinacea, quercetin, astralagus, lobelia, eucalyptus, and serrapeptase.

Even though herbs can have a significant role to play in the treatment of COPD, you should consult your doctor before starting the use of any herb. (1)

Foods Rich in Antioxidants: Enhance Your Body’s Immunity

Foods rich in antioxidants such as soy help to reduce the oxidative stress and chronic inflammation associated with COPD. Soy is a rich source of omega 3 fatty acids that work to minimize the incidences of breathlessness considerably because of its anti-inflammatory properties. It can also reduce the inflammation of airway in people with COPD.

Other rich sources of omega 3 fatty acids include canola oil, oily fish, flaxseeds and walnuts. You can use antioxidants, either in their natural form or as supplements for managing the symptoms of COPD. (2)

Natural Supplements: Fight Breathlessness Bravely

According to experts, natural supplements help to fight the symptoms of COPD effectively. Doctors have confirmed that one of the leading causes of COPD is a vitamin deficiency. Thus, taking vitamins such as C, D, or E helps to treat the condition. Similarly, lack of magnesium can cause breathing issues; therefore supplementing the body with the needed amounts of magnesium can improve lung function and capacity.

Other natural supplements include an essential amino acid, glutamine, and creatine. Creatine can also increase muscle mass and overall performance. (3)

Salt Therapy: Eradicates Toxins Effectively

Salt therapy is a pioneering treatment that has led to a considerable improvement in people with COPD. It helps to reduce the intensity and occurrence of coughing and promotes swift recovery.

The inhalation of salt aerosol eradicates toxins, minimizes inflammation, widens airways and expels mucus. In addition, it has an exceptional anti-bactericidal effect that reduces the chance of infection. (4)

Combat COPD with Natural Remedies!

While there are numerous types of treatment available, natural remedies for COPD have emerged as one of the most effective methods for treating the condition. They are reliable, fast acting and can be used with other alternative treatments to ensure effective recovery.

Sources: healthline.com|webmd.com|ncbi.nlm.nih.gov|cdc.gov/nchs/fastats/leading
Categories
DYSLEXIA

How Dyslexia is a Different Brain, Not a Disease

As a frame of reference, the act of reading is a human invention that’s only a few thousand years old. In Europe and the United States, mass literacy—meaning more than 50 percent of the population knows how to read—has only been around for about 150 years. In other areas of the world, such as most of Asia, the Middle East, and Africa, it’s much more recent.

THE READING BRAIN

Because the human brain doesn’t come already wired to read, there is no “reading center” of the brain and there are no “reading genes.” As Proust and the Squid author Maryanne Wolf writes, each individual brain must learn how to read on its own.

In learning to read, the brain performs an amazing feat: it creates a specialized circuit that’s just for reading, forging a new circuit by combining parts of the brain that were originally designed to serve other functions, such as retrieving names. This new “reading circuit” combines processes from different areas of the brain and then runs at a speed so fast it’s nearly automatic.

But not all brains forge a flowing reading circuit easily. This is the case with dyslexia. Rather than being a disease or a medical condition (the common misperception), dyslexia is a different brain organization—one in which the brain’s reading circuit has been disrupted or re-routed in at least one way, and sometimes in two or three ways. This re-routing slows down critical parts of the reading process:

  • attaching the right sound to a letter happens more slowly and
  • forming words or sentences takes longer, then comprehending what was just read also takes longer

Dyslexia can additionally affect memory, especially working memory, making it harder for students to remember what they just read, or directions and learning sequences. It’s important to note that dyslexia is NOT caused by visual problems, and it isn’t the flipping of letters, or reading letters backwards, or mispronouncing words—and it’s not related to motivation or intelligence. It’s merely the result of a brain with a different organization that makes reading and writing more difficult.

READING CIRCUIT ISSUES

In the dyslexic brain, the reading circuit can be interrupted in several areas and cause problematic development. Because each brain is unique, there isn’t a singular form of dyslexia, but there are common issues:

ISSUE #1: Phonemic Awareness — identifying individual units of sound is a major challenge in the dyslexic brain. There are 44 sounds, or phonemes, in the English language. The main problem for kids with dyslexia is the ability to notice and work with all the sounds, and then be able to match those sounds to the right letters. In younger children, this is why not being able to rhyme words is an important early sign of dyslexia—often dyslexic kids don’t recognize the ways two rhyming words sound alike. English is an irregular language so there are many different combinations of letters that match to phonemes, making reading and spelling really hard for kids with challenges working with and remembering speech sounds.

ISSUE #2: Fluency, or getting the reading circuit to work together quickly, is the second-biggest issue. Even when children can process all the phonemes, they can still have trouble associating the sounds with the right letters, perhaps due to the right hemisphere’s control over the left hemisphere-centered language processing. This makes reading even the simplest words very slow, and then automaticity doesn’t develop properly.

ISSUE #3: Comprehension is the third (but no less crucial) issue in reading. Once letters and sounds are matched together, some children will have trouble putting the words together to form sentences and meaning. Often, this kind of dyslexia doesn’t show up until children are older, around third grade and up, when there is a switch from learning to read to reading to learn.

For dyslexics, the work of reading is slow and laborious, so often otherwise bright and intelligent students who haven’t received proper intervention never become fluent readers. Many dyslexic students can get by for years by memorizing words without actually reading them, but by the third or fourth grade, when learning switches from learning to read to reading to learn, struggling students can’t read fast enough to keep up. They often experience repeated failure.

Yet, with explicit and systematic specialized instruction specifically for the different brain structure, the reading circuit can be re-trained to work toward fluidity. But first, students, parents, and teachers must understand that the dyslexic’s brain isn’t “broken” or deficient, just organized in a different way.

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